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  1. The Gs-Linked Receptor GPR3 Maintains Meiotic Arrest in Mammalian Oocytes (PDF)
  2. Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs(PDF)
  3. Oocyte-specific expression of Gpr3 is required for the maintenance of meiotic arrest in mouse oocytes
  4. Generation of mouse oocytes defective in cAMP synthesis and degradation: Endogenous cyclic AMP is essential for meiotic arrest (PDF)
  5. Mouse oocytes gradually develop the capacity for activation during the metaphase II arrest (PDF)
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  6. The Mos/mitogen-activated protein kinase (MAPK) pathway regulates the size and degradation of the first polar body in maturing mouse oocytes (PDF)
  7. The maternal nucleolus is essential for early embryonic development in mammals (PDF) Dodatkowe dane
  8. Strontium supports capacitation and the acrosome reaction in mouse sperm and rapidly activates mouse eggs (PDF)
  9. The exit of mouse oocytes from meiotic M-phase requires an intact spindle during intracellular calcium release (PDF)
  10. Transformation of sperm nuclei to metaphase chromosomes (PDF)
  1. Epigenetic modifications during oocyte growth correlates with extended parthenogenetic development in the mouse (PDF)
  2. Birth of parthenogenetic mice that can develop to adulthood (PDF) Komentarz Materiały dodatkowe
  3. Endogenous transcription occurs at the 1-cell stage in the mouse embryo (PDF)